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Existing research demonstrated that VB 12 administration significantly blunts the kidney damage caused by ischemia/reperfusion via marked suppression of ROS and associated inflammation and apoptotic cell death [14]

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33 When B 12 levels are too low in the body, the result is an increase in methylmalonic acid (MMA) and Hcy concentration due to inhibition of methylmalonyl-CoA mutase and methionine synthase, respectively