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In addition to SOD1 Cu metalation, in vitro data generated from hSOD1 G93A ‐transfected NSC‐34 cells implicates nitric oxide (NO) accumulation in SOD1‐associated mitochondrial dysfunction, [204] with substantial improvements in electron transport chain function elicited by NO scavengers
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Veterinary testing: Laboratory tests are performed to make sure either therapy is showing its effects or not
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