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These neuroregeneration studies have contributed to understanding of how actin dynamics-dependent cell migration and growth factor receptor-mediated cytoprotection interact in the complex biology of CNS repair , where both cell survival and the directed migration of progenitor and repair cells are essential for meaningful regeneration
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A metabolomics analysis of pancreatic ductal adenocarcinoma and thyroid cancer revealed that glutamine consumption was extremely high in tumor tissues compared with nontumor tissues [6]
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